Pathoanatomy Module 2 Pathoanatomy Module 2Please enter your email: 1. The forms of leukemia in children, with a favorable prognosis 1) T-lymphoblastic 2) B-lymphoblastic 3) myeloblastic 4) undifferentiated 5) plazmoblastic2. Macroscopic picture of subacute glomerulonephritis 1) great spotted kidney 2) a large white kidney greasy 3) primary contracted kidney 4) kidney with necrosis papillae of the pyramids3. The risk factors for atherosclerosis 1) stress 2) obesity 3) hyperuricemia 4) male 5) fermentopathy 6) hypercalcemia4. The relevant factor in the development of pyelonephritis 1) Megauretra 2) stricture of the urethra 3) purulent cystitis 4) chronic tonsillitis 5) sepsis5. TYPES lipoprotein metabolism disorders in atherosclerosis 1) reduction in LDL – cholesterol 2) increase the level of LDL – cholesterol 3) increase in HDL – cholesterol 4) improving the abnormal lipoprotein6. Histological forms of the PROSTATE CANCER 1) squamous 2) solid 3) high-grade carcinoma 4) anaplastic carcinoma 5) transitional cell7. Signs of acute lymphoblastic leukemia 1) The peak incidence at age 60 2) develops mainly in children 3) the prevalence of lymphoblasts in the bone marrow and blood 4) struck lymph nodes 5) in the leukemic infiltrates predominate myeloblasts8. Cirrhosis can be caused by 1) a fulminant hepatitis 2) diabetes 3) purulent osteomyelitis 4) the alimentary protein deficiency 5) alcoholism9. Morphological signs of portal cirrhosis 1) small tuberosity of liver 2) wide connective field 3) fine-meshed network of connective tissue in the lobules 4) early hepatic failure 5) early portal hypertension10. Histological signs of acute cholecystitis 1) neutrophilic infiltration of the bladder wall 2) sclerosis of the bladder wall 3) lymphoid infiltration of the bladder 4) necrosis of the bladder wall 5) imposition of fibrin on the mucous11. Trophoblastic disease 1) Vesical drift 2) placental transfusion syndrome 3) adenocarcinoma 4) invasive vesical drift 5) choriocarcinoma12. The stage of development of lobar pneumonia 1) gray hepatization 2) yellow dystrophy 3) mucoid swelling 4) congestion 5) Resolution 6) red hepatization13. A characteristic morphological features of focal pneumonia 1) involvement of the pleura 2) acute bronchitis and bronchiolitis 3) necrosis cheesy of exudate 4) fibrinous exudate in the lumen of the alveoli 5) defeat lobe14. forms of acute ischemic heart disease 1) cardiomyopathy 2) acute focal ischemic myocardium dystrophy 3) myocardial infarction 4) chronic cardiac aneurysm 5) sudden cardiac death15. Ischemic heart disease pathogenetic CONNECTION 1) with rheumatoid koronariitom 2) with mitral valve stenosis 3) with coronary atherosclerosis 4) with essential hypertension16. ischemic cerebral infarction develops in 1) vessels in the brain rupture of microaneurysms 2) thrombosis of cerebral arteries 3) thrombosis of the carotid and vertebral arteries17. Signs of chronic pyelonephritis 1) symmetrically evenly contracted 2) asymmetric uneven contracted 3) lymphohistiocytic infiltration, sclerosis of the stroma and periglomerular sclerosis 4) cystic tubular atrophy with the advent of lumens in their dense eosinophilic masses 5) glomerular sclerosis and hyalinosis.18. LOCATION IN characteristic changes at the membranous glomerulonephritis 1) proximal tubules 2) the distal tubules 3) the basement membrane of the tubules 4) basement membrane glomerular capillaries19. cardiomyopathy is characterized 1) valves lesion 2) Coronary thrombosis 3) focal granulomas in the myocardium 4) exudative interstitial inflammation 5) dystrophic cardiomyocytes20. In chronic leukemia is more characteristic 1) The proliferation of undifferentiated (blast) cells 2) the proliferation of ripening (cytic) cells21. Morphological signs of acute nephrosis 1) tubular atrophy 2) hyalinosis glomeruli 3) necrosis of the tubular epithelium 4) the formation of cylinders22. The most frequent localization of myocardial infarction 1) the right atrium 2) the left atrium 3) the right ventricle 4) the left ventricle23. SIGNS eccentric myocardial hypertrophy in hypertension 1) an increase in heart size in diameter 2) the expansion of adipose tissue 3) expansion of cavities 4) myocardial atrophy24. Complications of chronic pyelonephritis 1) Perinefrit 2) recovery 3) bleeding in the brain 4) uremia25. SIGNS OF THROMBOSIS of fetal ARTERY villi 1) fibrosis and obliteration of small vessels 2) fetal a bleeding disorder 3) bleeding in intervillous space 4) intervillous space free 5) the umbilical sepsis26. Bronchiectasis IT IS 1) expansion of the lumen of the alveoli 2) expansion of the lumen and increase the size of the bronchial glands 3) expansion of the bronchi 4) retention cyst27. Morphological manifestations of cerebrovascular Diseases 1) congenital aneurysm of the brain arteries 2) ischemic cerebral infarction 3) bleeding in the brain 4) Encephalitis28. The size of the heart in dilated cardiomyopathy 1) significantly reduced 2) slightly decreased 3) is not changed 4) slightly increased 5) increased significantly29. Bronchiectasis – a pathological EXPANSION 1) the lumen of the alveoli 2) one or more of the lumen of the bronchi, comprising cartilaginous plate and glands with destruction lamina propria and muscularis 3) small bronchi30. Histological forms of liver cancer 1) hepatocellular carcinoma 2) cholangiocellular cancer 3) anaplastic cancer 4) small cell cancer31. The important factor for developing chronic bronchitis 1) heart failure 2) Smoking 3) pulmonary hemosiderosis 4) lymphostasis 5) industrial dust32. pathogenesis of rheumatic diseases 1) immunodeficiency syndrome 2) violation of transplantation immunity 3) autoimmune reactions33. fatal complications in atherosclerosis mesenteric artery 1) intestinal obstruction 2) bowel gangrene 3) fibrinous colitis 4) ischemic colitis34. The conditions that predispose to DEVELOPMENT of nephrolithiasis 1) sickle cell nephropathy 2) hyperparathyroidism 3) gout 4) amyloid nephropathy 5) hyperoxaluria35. The cause of death of patients with cirrhosis 1) pulmonary embolism 2) hepatocellular insufficiency 3) complications of portal hypertension 4) hepatocellular carcinoma 5) secondary bacterial infection36. IMPROVING LDL in plasma leads to 1) the destruction of elastic fibers 2) increased permeability of the endothelium 3) damage and loss of endothelial cells 4) increasing adhesion of monocytes to Endothelial cells37. MOST PROVEN role in the development RHEUMATISM 1) beta-hemolytic streptococcus group A 2) beta-hemolytic streptococcus group B 3) Herpes Virus 4) kampillobakter38. Characteristic signs CROHN’S DISEASE 1) the place of defeat – rectum 2) chronic inflammation affects the entire thickness of the bowel 3) the mucous membrane looks like a cobblestone street 4) is characterized by crypt abscesses 5) characterized pseudopolyps 6) often leads to the development of colon cancer 7) is often complicated by fistulas interintestinal39. Histological forms of gallbladder cancer 1) scirrhus 2) adenocarcinoma 3) epidermal cancer 4) mucosal cancer40. Morphological basis of hepatosis 1) degeneration of hepatocytes 2) necrosis of hepatocytes 3) inflammation in the liver 4) pylephlebitis41. COMPLICATIONS OF GASTRIC CANCER 1) hemoptysis 2) dilation of the pylorus 3) perforation 4) depletion 5) gastric bleeding42. SIGNS hepatocellular insufficiency 1) hyperalbuminemia 2) icterus 3) encephalopathy 4) hepatorenal syndrome 5) coagulopathy43. The disease with development of primary nephrotic syndrome 1) renal amyloidosis 2) diabetic nephropathy 3) membranous nephropathy 4) focal segmental glomerular hyalinosis 5) lupus nephritis44. The disease with development of secondary nephrotic syndrome 1) renal amyloidosis 2) diabetic nephropathy 3) membranous nephropathy 4) focal segmental glomerular hyalinosis 5) lupus nephritis45. ADENOMA THIS IS 1) benign tumor of glandular epithelium 2) malignant tumor of glandular epithelium 3) epidermal cancer 4) malignant tumor of the transitional epithelium 5) benign tumor of squamous epithelium46. Obstructive pulmonary disease 1) asthma 2) chronic obstructive bronchitis 3) chronic obstructive pulmonary emphysema 4) bronchiectasis 5) chronic bronchiolitis47. Morphological signs of postnecrotic cirrhosis 1) the approach of portal triads with each other and the central veins 2) degeneration and necrosis of hepatocytes 3) lympho-macrophage infiltration 4) leukocyte infiltration 5) cholangitis, cholestasis48. PRIMARY cardiomyopathy 1) The hypertrophic form 2) Dilated 3) constrictive 4) canalicular 5) The restrictive49. CHANGES IN PRODUCTIVITY extracapillary glomerulonephritis 1) protein dystrophy tubular epithelium 2) necrosis of the glomerular capillary loops 3) fibrin in the lumen of the capsule glomerulus 4) proliferation nephrothelial and podocyte to form lunate 5) nodules Kimmelstilya-Wilson50. POSSIBLE COMPLICATIONS of chronic glomerulonephritis 1) anemia 2) chronic renal failure 3) hyperglycemic coma 4) brain hemorrhage 5) cardiovascular failure51. Benign tumors STOMACH 1) angiosarcoma 2) adenoma 3) leiomyoma 4) adenocarcinoma52. METAPHORICAL NAME THE HEART AT fibrinous pericarditis 1) “armored” 2) “muscatel” 3) “hairy” 4) “tiger” 5) “sago”53. AGENTS interstitial pneumonia 1) Streptococcus 2) pneumococcus 3) Staphylococcus aureus 4) viruses 5) mycoplasma54. Morphological changes lymph nodes in lymphogranulomatosis 1) amyloid deposition in the stroma 2) numerous cells Reed-Sternberg 3) cells of Hodgkin 4) necrosis, sclerosis 5) hyperplasia bright centers of follicles55. The disease characterized by the formation of glomerular lunate 1) membranous nephropathy 2) lipoid nephrosis 3) subacute glomerulonephritis 4) acute post-streptococcal glomerulonephritis56. Histological signs of chronic cholecystitis 1) atrophy of the mucosa 2) sclerosis of the bladder wall 3) lymphoid infiltration of the bladder 4) necrosis of the bladder wall 5) imposition of fibrin on the mucous57. Cause of infarction of internal organs in rheumatism 1) aneurysms of large vessels 2) thromboembolic complications 3) beta-hemolytic streptococcus58. endocarditis Libman – Sachs is typical for 1) Rheumatism 2) atherosclerosis 3) Ankylosing spondylitis 4) systemic lupus erythematosus59. Characteristic signs of chronic myeloid leukemia 1) pyoid bone marrow 2) axillary resorption and osteoporosis 3) intralobular leukemic infiltration of the liver 4) infiltration of the portal stroma leukemia cells 5) aleukemic leukemia60. REASON IS silicosis dust containing 1) silicon dioxide 2) the particles of coal 3) talc 4) silicates61. chronic nonspecific lung diseases 1) pneumonia 2) chronic obstructive pulmonary emphysema 3) chronic bronchitis 4) bronchiectasis 5) brown induration of lungs62. The most commonly histologic type of central lung cancer 1) adenocarcinoma 2) the bronchioles-alveolar 3) squamous 4) small-cell 5) large cell63. Sclerotic deformation of stomach is the outcome 1) catarrhal gastritis 2) diphtheritic gastritis 3) corrosivity gastritis 4) abscess gastritis64. Morphological characters intrauterine hypoxia 1) thrombotic complications 2) diapedetic hemorrhage and edema 3) aspiration of amniotic fluid 4) depression of the respiratory center of the brain 5) meconium in the amniotic fluid65. ELEMENTS sarcoid granulomas 1) hearth cheesy necrosis 2) Neutrophils 3) CD4 + T lymphocytes 4) fibroblasts 5) epithelioid cells66. renal size with hypertension 1) Increase 2) conventional 3) Reduce67. General factors of stone formation 1) violation of the osmotic pressure 2) violation of protein metabolism 3) violation of mineral metabolism 4) avitaminoses 5) increase in blood viscosity68. morphological characters DESTRUCTIVE of bronchiectasis 1) perifocal inflammation 2) stretching acini 3) carnification 4) the gap mezhalveolyarnyh partitions.69. Morphological forms of acute gastritis 1) fibrinous 2) atrophic 3) hypertrophic 4) catarrhal 5) Corrosive (necrotic)70. Clinical and morphological signs of chronic atrophic gastritis in the acute stage 1) occurs frequently in patients with alcoholism 2) the mucosa is not changed 3) diffuse lymphoid infiltration plasmocytic with considerable admixture of PMN 4) focuses pyloric and intestinal metaplasia 5) gastric hyperacidity71. The possibility of changes in the kidney with renal artery atherosclerosis 1) infarcts 2) amyloidosis 3) embolic purulent nephritis 4) hydronephrosis transformation72. stage during myocardial infarction 1) necrotic 2) Mixed 3) ischemic 4) Organization 5) Compensatory73. The cause of iron deficiency anemia in pregnant and nursing mothers 1) lack of receipt of exogenous iron 2) lack of synthesis of endogenous iron74. Anemia characterizes 1) reduction in the number of red blood cells 2) an increase in hemoglobin in the blood 3) decrease in circulating blood volume 4) decrease in hemoglobin, and often in the number of erythrocytes per unit volume of blood75. Microscopic characteristics scirrhous stomach cancer 1) atypical cells with large nuclei are arranged in groups 2) atypical cells form cancer 3) massive proliferation of connective tissue 4) the abundance of mucus in the lumen of the glands 5) atypical cancer cells do not form76. spleen weight increase greatly the in leukemia 1) acute 2) chronic77. Morphologically eclampsia is characterized 1) systemic fibrinoid necrosis of small vessels 2) disseminated thrombosis of small vessels 3) necrosis and hemorrhage in the organs 4) suppurative metastases 5) vomiting78. MECHANISM OF BLEEDING IN ULCER 1) arrosive 2) diapedetic 3) due to rupture of the vessel 4) as a result of purulent fusion79. Pulmonary complications of lobar pneumonia 1) Encephalitis 2) carnification 3) lung abscess 4) purulent mediastinitis 5) a pleura empyema 6) gangrene (wet)80. 1. The most frequent localization of ectopic pregnancy 1) ovaries 2) the fallopian tubes 3) abdomen 4) the cervix 5) vagina81. 11. BONE MARROW strikes PRIMARY 1) malignant lymphoma 2) in leukemia82. SIGNS of the Alport syndrome 1) hereditary disease 2) acquired disease 3) Glomerulopathy 4) tubulointerstinalnoe disease83. arteriolosclerosis hyaline vascular lumen 1) is narrowed 2) expanded 3) is not changed84. pathological process of disorganization of connective tissue rheumatism 1) Sclerosis 2) mucoid swelling 3) inflammatory reaction 4) metaplasia 5) fibrinoid changes85. 1. Diseases leading to the development of primarily contracted kidney 1) Glomerulonephritis 2) pyelonephritis 3) hypertension 4) Atherosclerosis86. BASE cerebrovascular disease is 1) syphilis, cerebrovascular 2) atherosclerosis of cerebral arteries 3) essential hypertension 4) Hydrocephalus 5) rheumatoid vasculitis87. Signs of chronic gastric ulcer during the exacerbation 1) the presence of fibrinopurulent exudate on the surface 2) scar tissue interrupts muscle membrane at different depths 3) endangitis 4) fibrinoid changes in vascular walls and in the bottom of ulcers 5) The deepest zone is located is represented by coarse fiber ulcer scar tissue88. Signs of acute alcoholic hepatitis 1) fatty degeneration of hepatocytes 2) leukocyte infiltration 3) the presence of cells Mallory 4) the formation of Councilman’s bodies 5) focal necrosis of individual hepatocytes89. The structure of typical rheumatoid granulomas 1) the focus of fibrinoid necrosis 2) focus caseation 3) macrophage cells Anichkova 4) focus liquefactive necrosis 5) reaction neutrophilic90. Morphological manifestations of mucoviscidosis 1) hemorrhagic syndrome 2) retention cysts of pancreas and other organs 3) secondary fibrosis 4) jaundice 5) cirrhosis91. Types of Angina pectoris 1) labile 2) stable 3) disappear 4) spastic 5) Prinzmetala 6) unstable92. Contributing factors in the development of pyelonephritis 1) obstruction of the urinary tract 2) vesicoureteral reflux 3) hypertension 4) Pregnancy 5) atherosclerosis 6) diabetes93. Do not look at treatment with cytostatics leukemic infiltrates STORED IN 1) heart 2) Kidney 3) brain 4) spleen94. Changes in the epithelium in chronic gastritis 1) atrophy 2) intestinal metaplasia 3) hyperplasia 4) dysplasia95. KIDNEY SYMPTOMS PYELONEPHRITIS 1) oligouriya 2) hematuria 3) leukocyturia 4) bacteriuria 5) dysuria 6) pain96. Changes arise in the kidney with chronic glomerulonephritis 1) thrombosis, necrosis of the glomerular loops 2) cell proliferation in renal corpuscles 3) glomerular sclerosis and hyalinosis 4) fibrinopurulent hemorrhagic exudate 5) infiltration in the stroma of histiocytes, and plasma cells97. Signs of chronic viral hepatitis “C” 1) macrovesicular steatosis of hepatocytes 2) the formation of lymphoid follicles in the portal tracts 3) confluent and bridging necrosis of hepatocytes 4) apoptotic Councilman’s bodies 5) expansion of portal tracts due to fibrosis98. Local factors in the development of gastric ulcer 1) increase the aggressiveness of gastric juice 2) campylobacter 3) presence of chronic gastritis 4) poor circulation 5) All the answers are correct 6) All the answers are incorrect99. carnification lungs at lobar pneumonia 1) the manifestation of the disease 2) complication of the disease 3) the outcome of the disease100. GROWTH IN NODES at the nodular prostatic hyperplasia starts at 1) The posterior lobe 2) preprostatic area 3) the prostatic urethra 4) anterior lobe101. The causes and risk factors of development of secondary hypertension 1) pheochromocytoma 2) coarctation of the aorta 3) violation of the separation of sodium by the kidneys 4) genetic disorders of the renin-angiotensin system102. pneumonia refers to groups dyscirculatory diseases the tumor disease inflammatory diseases processes of disregeneration All the answers are correct103. Stages of acute renal failure 1) uremic 2) shock 3) restoration of diuresis 4) nephrotic 5) oligoanuria104. Histological features of primary biliary cirrhosis 1) granulomatous cholangitis 2) decrease in the number of bile ducts 3) infiltration of portal tracts 4) expansion of portal tracts due to fibrosis105. Outcomes of toxic hepatic dystrophy 1) Portal cirrhosis 2) Postnecrotic cirrhosis 3)Biliary cirrhosis 4) Muscat cirrhosis106. SECONDARY biliary cirrhosis is characterized by 1) in the surface large hillocks 2) liver dark green color 3) bile stasis 4) in the surface small hillocks 5) associated with progressive massive hepatic necrosis 6) associated with obstruction of extrahepatic bile ducts107. The cause of development of ischemic colitis 1) Atherosclerosis mesenteric arteries 2) nonbacterial thrombotic endocarditis 3) the absence of ganglion cells in the submucosal layer 4) systemic vasculitis 5) Meckel’s diverticulum108. CAUSES OF ACUTE stomach ulcers 1) corticosteroids 2) Stress 3) Aspirin 4) Smoking 5) increasing vagal tone109. SIGNS OF INVASIVE vesical drift 1) lung metastases 2) tumor growth 3) transformation of cystic villi 4) the proliferation of trophoblast 5) swollen villi in the vessels of the myometrium110. Morphological changes of arterioles in hypertension 1) arteriolitis 2) caseous necrosis 3) fibrinoid necrosis 4) nodular periarteritis 5) hyaline arteriolosclerosis 6) hyperplastic arteriolosclerosis111. The primary lesion ORGANS in rheumatism 1) heart and blood vessels 2) the small joints 3) renal pelvis 4) these organs are not affected112. Outcomes on amyloid nephrosis 1) recovery 2) heart failure 3) uremia 4) death of autoinfection113. The forms of infectious endocarditis 1) acute 2) subacute 3) chronic 4) undulating114. Morphological signs of toxic hepatic dystrophy 1) reducing the size of the liver 2) imposing of fibrin on the capsule r 3) extensive necrosis of hepatocytes 4) grainy surface 5) increase the size of the liver 6) the flabby consistency of live115. Morphological signs of prematurity 1) the absence of ossification centers in the epiphysis 2) imperforate fontanelles 3) soft skull bones 4) the absence of vellus hair, face, shoulders, back, 5) underdevelopment of the nail plate116. CELLS Anichkova in rheumatoid granuloma BE CONSTRUED AS A 1) lymphoid cells 2) plump histiocytes 3) activated plasma cells 4) epithelioid cells117. successive stages of atherosclerosis 1) aterocalcinosis 2) fibrous plaques 3) complicated lesions 4) fatty Streaks 1) aterocalcinosis (4) 2) fibrous plaques (2) 3) complicated lesions (3) 4) fatty Streaks (1) 1) aterocalcinosis (2) 2) fibrous plaques (1) 3) complicated lesions (3) 4) fatty Streaks (4) 1) aterocalcinosis (2) 2) fibrous plaques (4) 3) complicated lesions (1) 4) fatty Streaks (3) 1) aterocalcinosis (4) 2) fibrous plaques (1) 3) complicated lesions (3) 4) fatty Streaks (2)118. Emphysema may be a manifestation 1) chronic nonspecific pulmonary inflammation 2) age-related processes 3) compensatory and adaptive processes 4) All the answers are correct 5) All the answers are incorrect119. The etiology of acute pyelonephritis 1) immune complexes 2) viruses 3) gram-negative bacteria 4) Gram-positive bacteria 5) hyperoxaluria120. Bence-Jones protein was detected in the urine at 1) chlamydia 2) Multiple Myeloma 3) chronic myeloid leukemia 4) chronic lymphocytic leukemia 6. spleen weight increase greatly121. morphological characters CROHN’S DISEASE 1) is characterized by segmental defeat cancer, “Hose stricture” 2) crypt abscesses 3) fibrinous plaques on the mucous membranes 4) atrophy of the mucosa 5) Noncaseating granulomas122. PRIMARY HEPATITIS 1) septic hepatitis 2) drug-induced hepatitis 3) alcoholic hepatitis 4) Viral Hepatitis 5) tuberculous hepatitis123. Causes of nutritional iron deficiency anemia 1) Gastrectomy 2) Pregnancy 3) autoimmune gastritis124. The form of clinical course of arterial hypertension 1) Secondary 2) idiopathic 3) malignant 4) benign125. Chronic viral hepatitis develop after 1) Hepatitis “B” 2) hepatitis “C” 3) Hepatitis “A” 4) the combined hepatitis “B” and «D» 5) Hepatitis “E”126. TYPE lymphogranulomatosis with the most unfavorable prognosis 1) predominance of lymphoid tissue 2) mixed cell 3) nodular sclerosis 4) Lymphocyte depletion 5) lymphohistiocytic127. CAUSE Crnification 1) enhanced fibrinolysis of exudate 2) joining pyogenic flora 3) insufficient fibrinolytic activity of leukocytes 4) the presence of extrapulmonary complications 5) All the answers are correct 6) All the answers are incorrect128. RHEUMATIC pancarditis this defeat 1) endocardium and myocardium 2) endocardial and pericardial 3) the myocardium and pericardium 4) endocardium, pericardium and myocardium129. Clue cells in lymphoid tissues IN lymphogranulomatosis 1) cells Anichkova 2) cells of Hodgkin 3) cells Mikulic 4) cells Reed-Sternberg130. Complications of chronic ulcers 1) penetration 2) perforations 3) empyema 4) hypercalcemia 5) scarry stenosis and deformation of the wall 6) bleed131. Signs of chronic persistent forms of viral hepatitis “B” 1) stored lobular structure 2) periportal fibrosis 3) infiltration in the portal tracts 4) expressed cholestasis 5) apoptotic Mallory’s bodies132. SIGNS dilated cardiomyopathy 1) dilatation of the heart chambers 2) dilation of the left ventricle 3) dilation of both atria 4) hypertrophy of the left ventricular wall 5) hypertrophy of the walls of the heart chambers 6) hypertrophy of the right ventricular wall133. Morphological signs of ulcerative colitis 1) ulcerative process within the mucosa 2) lymphoplasmacytic infiltrate all layers of the bowel wall 3) deep ulcers to the muscle layer 4) crypt abscesses 5) lymphoplasmacytic infiltration of the lamina propria with an admixture of eosinophils and leukocytes134. EXODUS rheumatic endocarditis 1) brown atrophy of the myocardium 2) the formation of heart disease 3) small-focal cardiosclerosis 4) carcinoid defeat valve135. Mönckeberg medial sclerosis strikes lining of arteries 1) Internal 2) Medium 3) outer136. The frequency of spontaneous abortion 1) 5-10% 2) 10-20% 3) 30-40% 4) 40-50% 5) 50%137. SIGNS OF infarction OF THE PLACENTA 1) a sharp narrowing of the intervillous space 2) narrowing of blood vessels in the villi 3) hemorrhagic impregnation of placenta 4) infiltration of polymorphonuclear leukocytes 5) calcification138. When atherosclerosis affects 1) veins 2) capillaries 3) arterioles 4) large and medium arteries139. Inflammatory reactions in rheumatism 1) purulent – exudative 2) predominantly alterative 3) intermediates 4) granulomatous140. Macroscopic picture of amyloidosis kidney 1) Great Spotted kidney 2) a large white kidney greasy 3) The big red kidney 4) Kidney with the foci of purulent inflammation141. CAUSES pneumoconiosis 1) industrial poisons 2) the effect of physical factors 3) Infection 4) industrial dust142. CHANGES cardiomyocytes of Angina pectoris 1) atrophy 2) kariolizis 3) fatty degeneration 4) the disappearance of glycogen143. The reasons for late GESTOSIS 1) violation of trophoblast invasion into the myometrium 2) cystic degeneration of placenta 3) The pathology of the uterine spiral arteries 4) calcification of the placenta 5) the allocation of the ischemic placenta tromboplastic substances144. Morphological characters lobar pneumonia AT THE STAGE OF GREY hepatization 1) fibrin in alveoli 2) the leukocytes and macrophages in the alveolar exudate 3) purulent exudate meltdown 4) edematous fluid in the alveoli 5) collapse of pulmonary capillaries145. The symptoms of acute catarrhal gastritis 1) mucosal thickening 2) atrophy of the glands 3) multiple erosions 4) mucous sclerosis 5) neutrophilic infiltration of the mucous 6) mucosal lymphoid infiltration146. The characteristic signs of ulcerative colitis 1) the place of defeat – rectum 2) chronic inflammation affects the entire thickness of the bowel 3) the mucous membrane looks like a cobblestone street 4) is characterized by crypt abscesses 5) characterized pseudopolyps 6) often leads to the development of colon cancer 7) is often complicated by fistulas interintestinal147. The changes in the lungs with diffuse chronic bronchitis 1) small focal atelectasis 2) macrofocal sclerosis 3) the formation of destructive bronchiectasis 4) a mesh fibrosis 5) carnification148. Pathological processes in the kidney with acute pyelonephritis 1) serous exudation into the lumen of the glomerular capsule 2) multiple abscesses 3) Fibropurulent a pyelitis 4) fibrinous exudate in the lumen of the glomerular capsule149. Carnification – IT IS 1) Organization of exudate in the alveoli with the formation of granulation tissue initially, and then the mature connective tissue 2) excessive activity of leukocytes in the exudate 3) suppuration of exudate 4) bleeding in the exudate150. AGENTS lobar pneumonia 1) pneumococcus 2) Staphylococcus aureus 3) Streptococcus 4) virus pneumotropic 5) Klebsiella151. GLOMERULONEPHRITIS CHARACTERISTIC 1) bilateral renal damage 2) the primary lesion glomeruli 3) unilateral renal damage 4) purulent inflammation 5) non purulent inflammation 6) predominant involvement of the interstitial tissue152. Morphological changes in ischemic colitis 1) hemorrhagic infarction 2) gangrene 3) megacolon 4) the proliferation of granulation tissue with subsequent fibrosis 5) polyposis mucosa153. CHANGES large vessels in essential hypertension 1) fat spots and stripes 2) hyalinosis walls 3) productive vasculitis 4) circular arteriosclerosis 5) aneurysm154. Macroscopic characteristics of nodes at the nodular prostatic hyperplasia 1) Yellow-pink 2) soft consistency 1) Yellow-pink 2) soft consistency 3) foci of hemorrhage on the cut 4) with the cut surface prostatic fluid flow down155. Complications of chronic obstructive bronchitis 1) cardiopulmonary failure 2) pneumonia 3) gangrene 4) pulmonary hemorrhage.156. The most frequently histologic type of peripheral lung cancer 1) adenocarcinoma 2) the bronchioles-alveolar 3) squamous 4) small-cell 5) large cell157. For lobar pneumonia is characterized by 1) defeat of an entire lobe or more lung lobes 2) purulent inflammatory exudate 3) fibrinous pleuritis 4) the gradual onset of the disease 5) the primary lesion of the bronchi158. Tubulointerstitial nephritis caused immune disorders and associated with antibody and glomeruli were observed at 1) syndrome Gudspachera 2) Albright syndrome 3) intoxication159. The outcome in necrotic nephrosis 1) recovery 2) acute renal failure 3) chronic renal failure 4) primary contracted kidney160. CAUSE hemolytic anemia 1) lack factor Castle 2) incompatible blood transfusion161. The TRUE NAME RHEUMATISM 1) Lyell’s disease (total cutaneous epidermolysis) 2) Buerger’s disease (systemic vasculitis) 3) parietal endocarditis with eosinophilia Loeffler 4) All the answers are correct 5) disease Sokolsky-Buyo162. macroscopic myocardial infarction revealed through 1) 1-2h 2) 4-6 h 3) 18-24h 4) 72 hours163. foam cells come from 1) macrophages 2) lymphocytes 3) mast cells 4) plasma cells 5) adventitial cells 6) smooth muscle cells164. CAUSES OF steatosis 1) poor quality food poisoning 2) Alcohol 3) mushroom poisoning 4) diabetes 5) viral hepatitis165. CAUSES PLACENTAL hematoma 1) thrombosis of the spiral arteries 2) infarction 3) placental abruption 4) breaking of spiral arteries 5) breaking of arterioles decidua166. The cause of development of appendicitis 1) blockage of coprolites 2) appendicular artery thrombosis 3) obstruction of gallstones 4) compression of the veins process 5) microbial flora167. warty endocarditis imposed on in rheumatism CONSTITUTE 1) Aschoff’s body 2) granulomas Berezovsky – Sternberg 3) imposition of thrombotic168. Morphological characters of Postmaturity 1) calcification of the placenta 2) absence of lubrication 3) dry skin with maceration 4) the appearance of ossification centers in the proximal epiphysis of the tibia and humerus 5) loss of the umbilical cord169. TYPES myocardial infarction localization 1) subendocardial 2) chordal 3) transmural 4) intramural 5) the valve 6) subepicardial170. microscopic features of myocardial infarction 1) plasma-coagulation 2) fatty degeneration 3) mucoid swelling 4) vacuolization of the cytoplasm 5) karyopyknosis, karyorrhexis171. Morphological signs of hemolytic disease 1) arteritis and phlebitis umbilical vessels 2) brown atrophy of the liver 3) hemolytic jaundice 4) anemia and edema 5) kernicterus172. The possibility of changes in the arterioles in essential hypertension 1) proliferation of endothelial 2) hyalinosis 3) giperelastoz 4) hypertrophy of muscle cells 5) fibrinoid necrosis173. Basic morphological signs of acute pyelonephritis 1) leukocyte infiltration interstitial 2) degenerative changes in tubular epithelium 3) protein cylinders in the tubules174. CELLS IN THE COMPOSITION rheumatic granuloma 1) Lymphocytes 2) macrophages 3) plasma cells 4) foam cells 5) fibrocytes175. Bladder tumors 1) papilloma 2) condyloma 3) adenocarcinoma 4) transitional cell cancer 5) leiomyoma176. TITLE rheumatic granuloma 1) focus Abrikosov 2) focus Aschoff Bullet 3) Aschoff’s body177. abdominal aortic aneurysm is typical for 1) Syphilis 2) Rheumatism 3) atherosclerosis 4) arteriosclerosis178. MAIN TYPES of bronchiectasis 1) varicose 2) Cylinder 3) mushroom 4) saccular 5) stellate179. The features typical of Burkitt’s lymphoma 1) a high degree of malignancy 2) is more common in people of Europe 3) the picture “starry sky” 4) marked cellular polymorphism of tumor tissue 5) detection of tumor cells virus Epstein – Bar180. 12. Bone marrow involvement lymphoma 1) secondary to metastatic 2) primary Loading …Is fire hot or cold?
