Pathophysiology Module 3 Mcqs

Pathophysiology Module 3 Mcqs

1. INADEQUATE (EXCESSIVE) PRODUCTION OF ANTIDIURETIC HORMONE WILL LEAD TO THE DEVELOPMENT OF

 
 
 

2. THE DEVELOPMENT OF PULMONARY EDEMA IS A RESULT OF THE FAILURE OF

 
 

3. THE COMPENSATORY MECHANISM OF RESPIRATORY ACIDOSIS IS

 
 
 
 
 
 

4. ALKALOSIS OCCURS DUE TO

 
 
 
 

5. Metabolic alkalosis is characterized by:

 
 

6. THE PRODUCTION OF ALDOSTERONE IN ADRENAL GLANDS IS STIMULATED BY

 
 
 
 
 
 
 

7. FOR ISOOSMOLAR REDUCTION OF THE TOTAL VOLUME OF EXTRACELLULAR FLUID ARE CHARACTERISTIC THE FOLLOWING SIGNS

 
 
 
 
 
 
 
 

8. ODEMAS AS A RESULT OF HEART FAILURE, RENAL FAILURE, LIVER CIRRHOSIS DEVELOP DUE TO

 
 
 

9. HYDROSTATIC PRESSURE IS LEADING FACTOR IN THE DEVELOPMENT OF FOLOWING EDEMAS

 
 
 
 

10. All of the following are compensatory mechanisms of metabolic acidosis, except:

 
 
 
 

11. DEVELOPMENT OF OSTEOPOROSIS OCCURS IN THE FOLLOWING CASES

 
 
 
 
 

12. Compensatory mechanism of isoosmolar hyperhydration is:

 
 
 
 

13. THE USE OF CARBONIC ANHYDRASE INHIBITORS MAY LEAD TO

 
 
 
 

14. The main factor of the development of allergic edema is:

 
 
 

15. pCO 2 IN THE BLOOD WILL DECREASE IN

 
 
 
 

16. THE USE OF «LOOP DIURETICS» MAY LEAD TO

 
 
 
 

17. HYPERCHLOREMIA IS TYPICAL FOR FOLLOWING DISORDER

 
 
 
 

18. The main factor of the development of nephrotic edema is:

 
 
 

19. THE PROXIMAL RENAL TUBULAR ACIDOSIS, «SALT- LOSING» KIDNEY AND DIURETICS LEADS TO DEVELOPMENT OF

 
 
 

20. THE CONCENTRATION OF HYDROGEN IONS IN ALKALOSIS

 
 
 

21. THE ALBUMIN-GLOBULIN INDEX OF PLASMA (WHEN TOTAL CONTENT OF PROTEIN IS NORMAL) FOR OCCURRENCE OF HYPOONKIA (DECREASE OF
ONCOTIC PRESSURE) MUST

 
 

22. THE MOST EFFECTIVE MECHANISM OF COMPENSATION IN ISOOSMOLAR HYPERHYDRATION IS

 
 
 
 

23. BLOOD LOSS LEADS TO THE DEVELOPMENT OF

 
 
 

24. pCO 2 IN THE BLOOD WILL INCREASE IN

 
 
 
 

25. HIDDEN (LATENT) TETANY OCCURS IN

 
 
 
 
 

26. THE LEADING FACTOR IN THE DEVELOPMENT OF ALLERGIC EDEMAS IS

 
 
 
 

27. MECHANISMS OF COMPENSATION IN ISOOSMOLAR DEHYDRATION ARE

 
 
 
 
 

28. ACIDOGENESIS IN RENAL TUBULES IN INCREASED PRODUCTION OF ALDOSTERONE

 
 
 

29. FOLLOWING PROCESSES PARTICIPATE IN COMPENSATED RESPIRATORY ACIDOSIS

 
 
 
 
 
 

30. MECHANISMS OF COMPENSATION IN HYPOOSMOLAR DEHYDRATION ARE

 
 
 
 

31. Respiratory alkalosis is characterized by:

 
 

32. THE REABSORPTION OF HCO 3 – IN KIDNEYS IN THE HYPOKALEMIA

 
 
 

33. THE ACTIVATION OF RENIN-ANGIOTENSIN-ALDOSTERONE SYSTEM IS LEADING FACTOR IN THE DEVELOPMENT OF FOLLOWING TYPES OF EDEMAS

 
 
 
 
 

34. THE LIQUID FILTRATION FROM VESSELS INTO TISSUES IS INCREASED DUE TO

 
 
 
 
 

35. THE TRIGGERING FACTOR IN THE DEVELOPMENT OF CONGESTIVE EDEMAS IS

 
 
 
 
 

36. THE REABSORPTION OF HCO 3 – IN KIDNEYS IS STIMULATED BY

 
 

37. PRIMARY CHANGES OF THE BICARBONATE BUFFER IN THE RESPIRATORY ALKALOSIS ARE DUE TO

 
 
 
 

38. THE MOST EFFECTIVE MECHANISM OF COMPENSATION IN PRIMARY HYPERALDOSTERONISM IS

 
 
 
 
 

39. THE LEADING FACTOR IN THE DEVELOPMENT OF INFLAMMATORY EDEMAS IS

 
 
 
 
 
 

40. THE BLOOD ONCOTIC PRESSURE DECREASES DUE TO

 
 
 
 
 

41. THE MAIN FACTOR IN THE DEVELOPMENT OF CARDIAC EDEMAS IS

 
 
 
 
 

42. CAUSES OF METABOLIC ALKALOSIS ARE

 
 
 
 
 
 
 
 

43. Hyper-K+emia is typical sign of the following disorder:

 
 

44. PRIMARY CHANGES OF THE BICARBONATE BUFFER IN THE RESPIRATORY ACIDOSIS ARE DUE TO

 
 
 
 

45. Compensatory mechanism of hyperosmolar hyperhydration is:

 
 
 
 

46. CAUSES OF RESPIRATORY ALKALOSIS ARE

 
 
 
 
 
 
 
 

47. THE MEMBRANE FACTOR PLAYS A KEY ROLE IN THE PATHOGENESIS OF FOLLOWING EDEMAS

 
 
 
 

48. THE CHLORIDE ION CONCENTRATION IN THE BLOOD IN RESPIRATORY ACIDOSIS

 
 
 

49. THE MOST TYPICAL LOCATION OF RENAL EDEMAS IS

 
 
 

50. THE BLOOD VOLUME DURING CARDIAC EDEMA

 
 
 

51. DECREASE OF THE BLOOD CONCOTIC PRESSURE IS LEADING FACTOR IN THE DEVELOPMENT OF FOLLOWING TYPES OF EDEMAS

 
 
 
 

52. MECHАNISMS OF COMPENSATION IN HYPEROSMOLAR DEHYDRATION ARE

 
 
 
 

53. THE INITIAL STAGE OF PULMONARY EDEMA LEADS TO

 
 
 
 

54. HYPERKALEMIA IS TYPICAL FOR FOLLOWING DISORDER

 
 

55. Hypo-K+emia is typical sign of the following disorder:

 
 

56. THE DECREASED SYNTHESIS OF ACIDS AND AMMONIUM IN RENAL TUBULES LEADS TO DEVELOPMENT OF

 
 
 
 

57. The main factor of the development of cardiac edema is:

 
 
 

58. HYPOCHLOREMIA IS TYPICAL FOR FOLLOWING DISORDER

 
 
 
 

59. The main mechanisms of edema development are:

 
 
 
 
 
 
 

60. PRIMARY CHANGES OF THE BICARBONATE BUFFER IN THE METABOLIC ACIDOSIS ARE DUE TO

 
 
 
 

61. THE INTRAVASCULAR VOLUME DURING DECREASE OF THE BLOOD COLLOID-OSMOTIC PRESSURE

 
 
 

62. THE CONCENTRATION OF HYDROGEN IONS IN ACIDOSIS

 
 
 

63. ACIDOSIS OCCURS DUE TO

 
 
 
 

64. METABOLIC ALKALOSIS DEVELOPS IN THE FOLLOWING CONDITIONS

 
 
 
 
 

65. THE COMPENSATORY MECHANISM OF RESPIRATORY ALKALOSIS IS

 
 
 
 

66. THE ONCOTIC FACTOR PLAYS A LEADING ROLE IN THE PATHOGENESIS OF FOLLOWING EDEMAS

 
 
 
 

67. THE LEADING FACTOR IN THE DEVELOPMENT OF TOXIC EDEMAS IS

 
 
 
 

68. THE RESORPTION FROM TISSUES INTO VESSELS IS DECREASED DUE TO

 
 
 
 
 

69. The development of respiratory alkalosis may be due to:

 
 

70. PRIMARY CHANGES OF THE BICARBONATE BUFFER IN THE METABOLIC ALKALOSIS ARE DUE TO

 
 
 
 

71. THE COMPENSATORY MECHANISM OF RESPIRATORY ALKALOSIS IS

 
 
 
 
 

72. HYPOKALEMIA IS TYPICAL FOR FOLLOWING DISORDER

 
 

73. THE SEVERE DIARRHEA, INCREASED SWEATING LEAD TO DEVELOPMENT OF

 
 
 

74. HYPO-ONCOTIC EDEMAS ARE

 
 
 
 
 
 

75. NEUROMUSCULAR EXCITABILITY IN DECOMPENSATED RESPIRATORY ALKALOSIS

 
 
 

76. METABOLIC ACIDOSIS DEVELOPS IN THE FOLLOWING CONDITIONS

 
 
 
 
 

77. THE TRIGGERING FACTOR IN THE DEVELOPMENT OF «HUNGRY» EDEMAS IS

 
 
 
 

78. Metabolic acidosis is characterized by:

 
 

79. THE LEADING FACTOR IN DEVELOPMENT OF NEPHRITIC EDEMA IS

 
 
 
 
 

80. INTRODUCTION OF BICARBONATE IN RESPIRATORY ACIDOSIS IS NECESSARY

 
 

81. CHANGES OF pH OF THE BLOOD IN THE DECOMPENSATED ACIDOSIS ARE CHARACTERISED BY

 
 
 

82. FOLLOWING PROCESSES PARTICIPATE IN COMPENSATED RESPIRATORY ALKALOSIS

 
 
 
 
 
 

83. THE DEVELOPMENT OF RESPIRATORY ALKALOSIS IS DUE TO

 
 
 

84. CHANGES OF pН OF THE BLOOD IN DECOMPENSATED ALKALOSIS INCLUDE

 
 
 

85. FACTORS WHICH LEAD TO DEVELOPMENT OF EDEMA ARE

 
 
 
 

86. EDEMAS OF EXTREMITIES DEVELOP AS A RESULT OF THE FAILURE OF

 
 

87. THE CHLORIDE ION CONCENTRATION IN THE BLOOD IN RESPIRATORY ALKALOSIS

 
 
 

88. Respiratory acidosis is characterized by:

 
 

89. DECREASE OF THE BLOOD HYDROSTATIC PRESSURE IS LEADING FACTOR IN THE DEVELOPMENT OF FOLLOWING TYPES OF EDEMAS

 
 
 
 

90. THE TRIGGERING FACTOR IN THE DEVELOPMENT OF «NEPHROTIC» EDEMAS IS

 
 
 
 

91. Compensatory mechanism of hypoosmolar dehydration is:

 
 
 

92. THE DEVELOPMENT OF RESPIRATORY ACIDOSIS IS DUE TO

 
 
 

93. The development of respiratory acidosis may be due to:

 
 


 

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